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Article in English | IMSEAR | ID: sea-88180

ABSTRACT

Neurological deficits can occur following snake bite. It is usually due to intracerebral haemorrhage or subarachnoid bleed as a result of depletion of clotting factors. A healthy 14-years-old male developed bilateral ptosis and altered sensorium within 3 hours of snake bite. CT Brain revealed bilateral cerebellar and right occipital infarction with mass effect. Clotting time and bleeding time were normal. The possible mechanism for infarction in this patient is discussed.


Subject(s)
Adolescent , Anti-Bacterial Agents/therapeutic use , Aspirin/therapeutic use , Blepharoptosis/etiology , Diuretics/therapeutic use , Diuretics, Osmotic/therapeutic use , Fatal Outcome , Furosemide/therapeutic use , Humans , Infarction, Posterior Cerebral Artery/diagnosis , Male , Mannitol/therapeutic use , Occipital Lobe/pathology , Platelet Aggregation Inhibitors/therapeutic use , Risk Factors , Snake Bites/complications , Sodium Potassium Chloride Symporter Inhibitors/therapeutic use
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